Lyssavirus RBP121N | Characters

Lyssavirus RBP121N | Characters

 Gender: RNP->mRNA
 Height: 190 nm x 75 nm
 Created: November 6th, 2005
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Biography

The Rabies Virus
Rhabdoviridae Lyssavirus RBP121N (Rabies Brain Protocol 121st strain, N root)

A strain of rabies developed in the festering sewage of Void\'s few water purification plants, spread initially by rats and small rodents resident in the area where it quickly mutated into RBP001N-RBP120N. A final raccoon, dying, made its way to the void city zoo and perished feverishly in a pool of drinking water, where the virus underwent a final mutation via the water\'s tepid temperature and antibiotics/flouride content pumped into the zoo\'s water supply.

RBP121N\'s unique genetic code allows it to adapt to almost any warm-blooded mammal\'s DNA for replication, where it quickly mutates into a unique strain specific to that animal. The brain is infected very quickly, within several days of transmission, which is conducted through the exchange of bodily fluids, including saliva. Transmission is not aerosol. On infection it migrates with uncanny speed to the brain. Normal rabies symptoms will appear within four to twelve weeks of having been bitten, but RBP121N manages to directly target the brain within 12 hours. It is programmed to survive longer than other strains due to its slower infection rate once situated in the brain. The hypothalamus is attacked first, leaving the glands of the body haywire. Norepinephrine (noradrenaline) output is always at maximum, creating a very exhausted but alert and ready state for the host with maximum muscle and reflex output after the initial fatigue is hurdled. After this stage of infection, the rabies sets to work \"planning\" its next transmission.

Its only goal is to replicate. This is accomplished by RNA coding in the rabies designed to produce mass amounts of proteins which modify the \"feed\" muscle memory area of the basalganglia (including the hippocampus, which makes the virus\'s orders the first priority to the host), inducing the host to bite.

[I\'m not making most of this up, here\'s a quote from the September 6th 2005 New York Times science section regarding microbial and small-organism parasites:

...\"We found the parasite produces and injects proteins into the brain of its host,\" Dr. Thomas said.

Two of the proteins belonged to a well known family of signaling agents known as the Wnt family that are deployed in developing the cells of the nervous system.

Though produced by the worm [the article is about parasitic worms who induce grasshoppers to commit suicide by jumping into water, where the worm can exit the host body and spawn], the two proteins seemed similar to insect-type proteins and perhaps developed so as to mimic them, the French biologists report in an article in the current issue of Proceedings of the Royal Society.

Parasites have long been known to influence their hosts\' behavior in ways beneficial to the parasite. The rabies virus, for instance, makes animals rabid so that they bite others and transmit the virus.

An unusually specific instance of behavioral manipulation was discovered recently in a wasp that parasitizes an orb-weaving spider in Costa Rica.

The night before the wasp larva kills its host, it somehow reprograms the spider\'s web-building activity so that instead of its usual temporary web, the spider constructs a durable platform ideal for the larva to pupate on.

Somehow the larva reprograms the spider into executing, over and over again, just the first two steps in a five-step subroutine from the early phase of web-building.

If the larva is removed just before it can kill its host, the orb weaver will spin a platform-style web that and the following night, but revert to its usual web on the third night, as if it has shaken off some mesmerizing chemical the wasp has injected into its nervous system.]

The unique strain of RBP121N does incredible rearranging to this section of the brain. The host will stop at nothing to maneuver up to and bite its target. If the host cannot realize it is infected and obtain 1. a proprietary rabies immunoglobulin injection and 2. a vaccine within a week of this secondary stage of infection, the immune reaction, having already made the host feverish and ill, will incubate the virus to dangerous levels where rampant brain reprogramming will destroy the original brain structure, eradicating the memories and the personality of the host. The virus will continue to reproduce under feverish conditions for several weeks with the host at its command, for the personality/memory centers of the brain have already been destroyed. Eventually, fever temperatures and an overabundance of the RNP released by the virus will destroy both the virus and the basalganglia of the host. Death quickly follows.

Suffering a bite at the jaws of the host, it is up to the challenger to both realize the infection state within a week of infection and decide how to get rid of the rabies virus before his or her time is up! Created and spread throughout the zoo, there is no telling which kind of animal(s) the rabies virus has infected, or which kind the challenger will meet.

The brain of the host animal is usually beyond hope by the time it actively seeks contact with any one individual. The trait of foaming at the mouth comes from masticating the air continuously, creating a slight foam of saliva. Very often the foam is unnoticable--a much more telling trait is abnormal animal behavior. The animal will behave strangely, often operating outside of its usual circadian cycle. Rabies sufferers cannot sleep. This strain of rabies, as stated above, makes the animal behavior extremely aggressive in order to achieve the proximity required for a transmitting bite in this city of supernatural phenomena.



Infected by rabies?

1. Know your weight! The immunoglobulin injection must match your bodyweight. As much as possible but not more than necessary is injected around the bite site. Any extra that cannot saturate the bitten tissue must be injected into another area on the body.

2. The subsequent vaccine must be administered over the next week, comprising of 5 injections of 1cc each. Usually this schedule is spread out over 28 days, but due to the quick nature of RBP121N\'s reprogramming there is no choice for the host but to tax the already-impaired immune system slightly.

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